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Pathophysiology of of hemochromatosis

June 22, 2017 mfadmin Medicine

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Pathophysiology of of hemochromatosis

AR Excess iron absorption leading to fibrosis in involved organs secondary to hydroxyl free radicals

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Previous Post:Wilson disease,”AR mutation in ATP7B Impaired copper excretion into bile and incorporation of Cu into ceruloplasminCu accumulates in hepatocytes and kills them then links into various organs –> cornea, brain, kidney Labs: decreased ceruloplasminTreatment: d-penicillamine, zinc (prevents Cu uptake)

Next Post:Complications of hemochromatosis,”CirrhosisCardiomyopathyDMArthritis of 2nd and 3rd MCP

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